This is one of the best staples in my regime4g huperzine.
for concentration and motivation.
Any effects on mood, I can easily
counteract it with Ashwagandha
or even a cup of coffee.
Great to mix with Pine Bark
for an added mental kick and proper dosing.
My concentration improves some,
and my recall / short term is always 100% sharper.
MAO-A can selectively metabolize. lef member suggests tribestan .
adrenaline, noradrenaline, serotonin,
and dopamine to inactive compounds.
Tribulus contains Harmine,
a short-acting, "reversible" MAOI-A inhibitor
The inhibition of MAO-A prevents the degradation of
adrenaline, noradrenaline, serotonin, and dopamine
and the result can be increased mood,
alleviation of depression and more energy.
High dopamine levels can stimulate the pituitary gland
to release lutenizing hormone (LH)
which leads to an increase in testosterone levels
and thus elevating libido, another mood enhancer.
Harmine is "reversible" because it
does not bind permanently to MAO-A,
thus is not as contraindicated with
tyramine-containing foods (cheese, red wine, etc)
as irreversible MAOI's,
nor does harmine raise levels of tyramine per se
since it allows tyramine to be metabolized.
The short-acting and non-permanency
of it's binding capabilities means that
the anti-depressive effects of harmine
usually cease 24-48 hours after stopping therapy.
. with a half-life of 2.5 hours,
several doses are necessary;
eg 3 * 750 ... 1,500 mg
of standardized 45% steroidal sapponins.
. huge piles of wood will spontaniously[@] www.nrbp.org/papers/055.pdf
combust within 6months
if not turned over to remove
anaerobic decomposition
-- just like smaller piles of dung
that do the same much sooner .
Professor of Human Genetics
at the University of Oxford since 1997,
Bryan Sykes, discussed his DNA research ,..
Sykes described his work on the
"Seven Daughters of Eve."
The Y (male) chromosome in general has been subject to
more degeneration and mutation than the X (female) chromosome,
and, he warned, if this continues
males might eventually become extinct .
. y-chromosome holding male-ness is decomposing;
we could move male genes to another chromosome,
or we could just clone without males .
[ Primate Y chromosomes, including in humans,Criticism of the book and theory at Discover Magazine/
have degenerated so much
that primates will also evolve relatively soon,
new sex determination systems
in about 14 million years in humans.[7][25] ]
Bryan Sykes heavily invested,[. that post is not archived, but there is
both academically and commercially,
in his vision of The Seven Daughters of Eve (2001)
– the matriarchs of the European population,
only one of whom (Jasmine i.e. mtDNA J)
he pictured as arriving from the Near East with farming.
The rest he saw as more ancient in Europe.
Unfortunately for Sykes, he was just plain wrong.
Now that aDNA researchers have better techniques
to guard against contamination,
and can test to a much higher degree of
resolution on the mtDNA tree,
the picture has turned completely around.
Two papers last year showed that
Neolithic people in Northern Europe
were completely unrelated to their Mesolithic predecessors,
who were overwhelmingly U4 and U5.
New papers attempting to set their own material in context
do not always reflect the latest discoveries.
There is often a long delay between
submission and publication of a paper.
This is the second paper which has attempted to
reconcile the old Sykes story with
what is emerging from Y-DNA,
not realizing that the Seven Daughters saga
has gone the way of the dodo.
Some of the mtDNA haplogroups found in this study
are indeed ancient in Europe – the U5 for sure.
But not the majority.
.
It no longer looks as though H1 and H3 were
spread from the Franco-Cantabrian refuge
either. See my blog post:
Franco-Cantabrian refuge for what? Not mtDNA H1, H3.)
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