2023.1.29: web.cook/lentil/cooking time in a pressure cooker:
I reduce cooking time in 3 ways:
. soak the lentils in boiling water to soften them.
. puree the soaked lentils in a vitamixer;
and, that way some of the heat that was needed for
breaking up the lentils
can then instead be used for immediately
attacking anti-nutrients.
. use a high temperature by using an
InstantPot pressure cooker;
"Lima beans, Lentils, Peas;wiki:
White navy beans, properly prepared" .
. navy beans have a high saponin content;
Saponins exhibit antibacterial and anti-fungal activity,
and have been found to inhibit cancer cell growth.
. navy bean is the richest source of
ferulic acid [antibacterial] and p-coumaric acid
among the common bean varieties.
White beans are the most abundant plant-based source of
phosphatidylserine (PS) yet known .
Mannose can be found in berries,
green beans, eggplant, cabbage,
turnips, kelp and Aloe vera.
. Mannose [that exists on the surfaces of your cells]
plays a role in the formation of
cytokines which stimulate the immune system .
D-mannose blocks the lectins in legumes
and can prevent them from breaching the intestinal wall.
D-mannose can bind to things that cause
inflammation, irritable bowel syndrome,
weight gain and even possibly colon cancer.
Being Inactive Kills as Many as Cigarettesme:
- Longevity Center - Everyday Health
How about some truth in headlines:
Being a [hyper-glycemic dieter] May Kill You .
-- The Truth About Sex via The Truth About Sex
"removal of physical inactivity. it just so happens that beans are
had the largest effect on colon cancer,
and the smallest on coronary heart disease,
in terms of percentage reduction, ..."
. the participants were lightly active, lean,
and then drinking soft drinks for only four weeks.
. Dr. Hans-Peter Kubis, Bangor University in England .
. the research showed regularly drinking soft drinks
changed the way muscles use food as fuel,
making them prefer to burn sugars over fats
via altered gene activity
-- encouraging unhealthy adaptations
similar to those seen in people with
obesity problems and type 2 diabetes:
a reduced ability to burn fat and to fat gain.
and making it more difficult for the body to
cope with rises in blood sugar.
lean meat (skinless turkey and chicken, low-fat beef),me:
fish, vegetables, fruits, and nuts.
High protein, low carb, low fat.[/quote]
8.10: news.health/zone/dr.sears
intro
. this study indicates that diet
plays a considerable role in
the risk of [needing and being eligible for]
cataract extraction
in at least a certain Italian population,
with a protective action played by
some vegetables, fruit, calcium, folic acid,and vitamin E,
and an increased risk
associated with elevated salt and fat intake [6.29:
-- this could have been a measure of
where these Italians were getting their
extra doses of salt and fat:
the usual culprit is luncheon meat
-- known to promote diabetes by
mixing sugar with the meat .]
. a significant inverse trend in risk,
were found for intake of
meat (OR 0.6, 95% CI 0.4 to 0.9),
cheese (OR 0.7, 95% CI 0.5 to 1.0),
[implied a more low-carb' diet?
the atkins effect .
6.19: ie,
not necessarily an endorsement
of cheese ! ]
cruciferae (OR 0.5, 95% CI 0.3 to 0.8),
spinach (OR 0.6, 95% CI 0.4 to 0.9),
tomatoes (OR 0.5, 95% CI 0.4 to 0.8),
peppers (OR 0.7, 95% CI 0.4 to 1.1),
citrus fruit (OR 0.5, 95% CI 0.2 to 1.3),
and melon (OR 0.5, 95% CI 0.4 to 0.8).
. a significant increase in risk
was found for
the highest intake of
(compared to the lowest intake)
butter (OR 2.8, 95% CI 1.2 to 6.4),
[6.29: apparently Italian cheese is
not a significant source of butter ? ]
total fat(OR 1.8, 95% CI 1.2 to 2.8),
[ie, sat'fats vs oils]
salt (OR 2.4, 95% CI 1.4 to 4.0)
[luncheon meats vs fresh meats]
. a significant increase in risk
was found for consumption of
oil other than olive oil
[mono'fats vs poly'fats]
(OR 1.6, 95% CI 1.1 to 2.2).
Among micronutrients,
lower ORs for cataract extraction
(highest quintile of intake compared to the lowest)
were found for intake of
calcium (OR 0.5, 95% CI 0.3 to 0.8),
folic acid (OR 0.4, 95% CI 0.2 to 0.7),
and vitamin E (OR 0.5, 95% CI 0.3 to 1.0),
[this may have more to do with
the foods that provided these micronutrients]
. intakes of
methionine, retinol, beta-carotene,
and vitamins A, C, and D
were not associated with cataract risk .
. most of the dark green leafy vegetables,[6.20:
have 15-47% of their carotenoids as lutein,
but very little zeaxanthin (0-3%)
while most of the absorbed carotenoid[6.21:
is carried in LDL,
53% of the (lutein, zeaxanthins) group
is carried by HDL .
. melatonin stimulates glutathione production;[6.21:
it is a potent inhibitor of cataracts;
proven in animal models .
Melatonin production slows after age 40,
but by age 60 virtually no melatonin is produced
at a time when most cataracts develop.
. 3 mg of mel at bedtime in elders with AMD
prevented further vision loss (Yi 2005).
. ala prevents cataract by[. the Vitamin Book said vit'E's job was
protecting glutathione synthesis.
. in animal models it reduced cataract by 40%
and protected the lens from losing
vitamins C, E, and glutathione.
. vitamin C is a first-line neutralizer of radical;
it is then recycled by glutathione .
. Selenium works with alpha-lipoic acid
to increase cellular concentrations of glutathione,
. Low plasma levels of vitamin E
increase the risk of lens opacities.
. cysteine is a component of glutathione;50 .. 150 mg riboflavin:
A combination of diallyl disulfide
(a major organosulfide in garlic oil)
and N-acetyl-cysteine (NAC)
completely prevented cataract development in animals.
Riboflavin reduces oxidized glutathione;250 mg Inositol nicotinate:
it is a precursor FADH
(reduced flavin adenine dinucleotide)
which is used by glutathione reductase,
to reduce and activate glutathione;
it makes glutathione available for the enzyme
glutathione-selenium peroxidase,
which neutralizes peroxide free radicals .
. occurs in high concentrations in the lens.2.5 mg folic acid
Inositol quenches reactive oxygen .
. B-6 prevents free-radicals by100-200 mg CoQ10;
properly metabolizing homocysteine
and is required for absorption of vitamin B12,
and proper synthesis of nucleic acids.
. . low levels of the B vitamins that are
critical to the metabolism of homocysteine),
will result in elevated levels of homocysteine,
which is a risk factor for both AMD and
other cardiovascular diseases .
. supplementing with folic acid, B6, and B12
can significantly reduce the risk of AMD
in adults with high homocysteine (Christen 2009).
. Mitochondrial dysfunction can result inDHEA
reactive oxygen species
linked to both cataracts
and macular degeneration;
a combination of CoQ10, acetyl-L-carnitine,
and proper diet,
improved mitochondrial function .
Acetyl-L-carnitine can reduce cataract risk
by acetylating potential glycation sites
that would otherwise cloud lens crystallins .
. abnormally low in patients with AMD (Bucolo 2005).[6.21:
DHEA also has been shown to protect the eyes against
oxidative damage (Tamer 2007).
Gingko biloba has anti-cataract potential;500-1000 mg carnosine
Bilberry, a proanthocyanidin,
Gingko biloba and bilberry may restore
microcapillary circulation.
. bilberry contains Anthocyanidins
and Cyanidin-3-Glucoside (C3G);
. anthocyanidins may prevent leakage from capillaries,
prevalent in neovascular AMD.
Studies also show that bilberry increases
oxidative stress defense mechanisms in the eyes (Milbury 2007).
C3G fights inflammation by
inhibiting inducible nitric oxide synthase (iNOS),
C3G increases cytokine adiponectin
[. helps burn energy instead of letting it
raise sugar levels and increase fat .]
C3G may be protect against Alzheimer’s
[. that wouldn't be surprising,
if it does reduce sugar spiking .]
C3G helps to induce apoptosis
in a number of human cancer lines .
Bioflavonoids are powerful inhibitors of
the enzyme aldose reductase.
If aldose reductase activity falls,
sorbitol is not synthesized.
This reduces the accumulation of
water in the lens.
The bioflavonoids quercetin, myrcetin,
and kaempferol (from limes)
specifically inhibit diabetic cataracts.
Gingko is a widely used flavonoid that
maintains microcirculation to the eye
and inhibits free radicals.
inhibits formation of AGEsEye drops containing N-acetyl-L-carnosine
(advanced glycation end products)
can delay vision senescence in humans:Phil Micans PharmB, Vice President, IAS Group:
effective in 100% of primary senile cataract cases
and 80% of mature senile cataract cases.
N-acetyl-L-carnosine eye drops are approved for
human use in Russia for the treatment of many eye diseases.
Brite Eyes II contains
1% N-acetyl-L-carnosine
L-Carnosine is an anti-glycation agent;
Topically applied N-acetyl-carnosine
prevented light-induced DNA strand breaks
and repaired damaged DNA strands (Specht 2000),
and has improved visual acuity,
glare and lens opacification
in animals and humans with advanced cataracts
(Williams 2006, Babizhayez 2009).
. the application of L-carnosine for the treatment ofupdate understanding of cataract:
human cataracts is misleading.
This is because L-carnosine readily becomes a substrate for
the activity of natural peptidases (i.e. carnosinase)
in the aqueous humor.
So much so, that there is
no sign of L-carnosine in the aqueous humor
within 15 minutes after instillation.
Furthermore,
I consider that L-carnosine eye-drops
may even be harmful for eyes because
it gradually releases histamine,
which, located as it would be
in the presence of the eye-lens
is a very toxic agent.
However, NAC eye-drops are resistant to
hydrolysis with natural carnosinase.
Therefore, NAC is the only currently known agent
which reverses and prevents human cataracts.
"The antioxidant activity of L-carnosine (beta-alanyl-L-histidine,wrote M.A. Babizhayev and colleagues.
bioactivated in ocular tissues)
versus N-acetylcarnosine (N-acetyl-beta-alanyl-L-histidine,
ocular-targeted small dipeptide molecules)
was studied in aqueous solution
and in a lipid environment,
employing liposomes as a model of lipid membranes.
Reactive oxygen species (ROS) were generated by an
iron/ascorbate promoter system for induction of lipid peroxidation (LPO),"
investigators in Russia report .
"L-carnosine, which is stabilized from enzymatic hydrolysis,
operates as a universal aldehyde and ROS scavenger
in both aqueous and lipid environments
and is effective at preventing ROS-induced damage
to biomolecules.
Second-generation carnosine analogs bearing the
histidyl-hydrazide moiety
were synthesized and tested versus L-camosine
for their ability to reverse the glycation process,
also known as the Maillard reaction,
and reverse the stable intermolecular cross-links,
monitored in the glucose-ethylamine Schiff base model,
ultimately resulting in the formation of
the advanced glycation end products (AGES)
from nonenzymatic glycation,
accumulating in numerous body tissues and fluids.
The obtained data demonstrate the transglycation properties
of the ophthalmically stabilized L-camosine
and L-carnosine histidyl-hydrazide derivatives tested
and can be used to decrease or predict the occurrence of
long-term complications of AGE formation
and improve therapeutically the quality of vision
and length of life for diabetes mellitus patients
and survivors with early aging.
Scientists at Innovative Vision Products, Inc. (IVP),
developed lubricant eyedrops designed as a
sustained-release 1% N-acetylcarnosine prodrug of L-camosine.
The eyedrops contain a mucoadhesive cellulose-based compound
combined with corneal absorption promoters
and glycerine in a drug-delivery system.
Anti-aging therapeutics with the
eyedrop formula including N-acetylcarnosine
showed efficacy in the nonsurgical treatment of
age-related cataracts for enrolled participants
in the prospective, randomized, double-masked,
placebo-controlled crossover clinical trial
after controlling for age, gender, and daily activities.
In a cohort in excess of 50,500 various patients
seeking cutting-edge medical care,
the N-acetylcamosine topical eyedrops target therapy
was demonstrated to have significant efficacy, safety,
and good tolerability for the prevention and treatment of
visual impairment in this older population
with relatively stable patterns of causes
for blindness and visual impairment.
Overall, accumulated study data demonstrate that
the IVP-designed new vision-saving drugs,
including N-acetylcarnosine eyedrops,
promote health vision and prevent vision disability from
senile cataracts, primary open-angle glaucoma,
age-related macular degeneration, diabetic retinopathy,
and aging.
N-acetylcarnosine eyedrop therapy is the crown jewel of the
anti-aging medical movement
and revolutionizes early detection, treatment,
and rejuvenation of aging-related eye-disabling disorders,"
Epidemiological reviews have suggestedref# Exposure to phenothiazine drugs and risk of cataract.
a possible link between cataract occurrence
and other psychotropic medications,
including benzodiazepines .
. long-term use of corticosteroids, phenothiazines,
certain other antipsychotic drugs,
some antineoplastic agents, and amiodarone
may increase cataract incidence.
. in the mid-1960s,
a decade after the marketing of phenothiazines,
drug-related ocular changes were reported in
patients who had been receiving years of
high-dose antipsychotic medication;
chlorpromazine was the primary offender.
Research also associated cataract occurrence with
other phenothiazines;
thioridazine, thiothixene,
trifluoperazine, levomepromazine,
and perphenazine are frequently cited examples.
A 1999 study of cataract occurrence
compared schizophrenic patients (26%)
with the general population (<1%).
. in addition to pharmaceutical exposures
schiz'ics often have additional risk factors:
smoking and poor dietary habits,
along with emotion-based metabolic disorders
(high cortisol, high insulin,
and blood sugar instabilities) .
[6.21:
. some of the new atypicals are clearly
increasing carb' intolerance,
and thus any cataracts blamed on sugar instabilities
must again point the blame at these meds .]
Epidemiological reviews have suggested
a possible link between cataract occurrence
and other psychotropic medications,
including benzodiazepines
and certain antidepressants
of the tricyclic and mao inhibitor classes .
Clinical reports have indicated an increased risk ofref# Sunlight and other risk factors for cataracts:
ocular opacities in users of phenothiazine drugs,
and some recent epidemiologic studies have found
an association between cataract and a
history of tranquilizer use.
To examine the effects of major tranquilizers
(phenothiazines and haloperidol)
on the risk of cataract extraction,
while controlling for suspected risk factors
such as diabetes and steroid use,
a matched cohort study was performed .
The use of either antipsychotic
or other phenothiazine drugs
increased the risk of cataract extraction
by roughly 3.5 times
in those who were both current users
and were exposed some time in the
2 to 5 years prior to their extraction.
Risk was also increased in individuals with
prior use of
antidiabetic agents,
systemic steroids,
and benzodiazepines.
. relation between individual exposure to sunlightrenewed interest in issue with anti-aggressives
and the risk of cataracts.
Sunlight exposure was very slightly related to
all types of opacities combined.
Although the numbers of cases with
each type of opacity was small,
the risk of cataracts was slightly increased
in medium and high exposure categories
for persons having cortical or
posterior subcapsular opacities only,
but not nuclear sclerotic changes.
Persons with dark brown or hazel eyes
are at increased risk.
An unexpected finding was that
persons who reported using tranquilizers
for six months were at increased risk.
. recent questions about quetiapine (seroquel)-- agingeye.net's summary:
as a possible cataract inducer
have increased the current interest in this issue
across the antipsychotic pharmacotherapy spectrum.
. photosensitizing agents
like chlorpromazine and its metabolites
denature proteins,
which become opacified when exposed to sunlight
and are deposited in the lens, cornea, and skin.
Melanin is also considered a probable cause
of lens discoloration
because it traps free radicals produced by
chlorpromazine.
Melanin and chlorpromazine
have a strong bonding affinity.
When chlorpromazine interacts with
ultraviolet-B light,
it produces purple or bluish colored
free-radical metabolites.
This phototoxic reaction creates the
cataract cellular changes.
has also been reported to induce[6.29:
lenticular changes unrelated to pigmentary retinopathy.
[Siddall J:
Ocular complications related to phenothiazines.
Dis Nerv Syst 1968; 29:10-13]
However, contrary reports have indicated
no evidence of lens opacity
in patients taking this drug.
[Barnes G, Cameron M:
Skin and eye changes with chlorpromazine therapy.
Med J Aust 1966; 1:478-481]
the risk of cataract occurrence remains unclear.
Cataract occurrence was observed as an infrequent finding6.22:
during screening for adverse events
before ziprasidone was approved for clinical use.
[Geodon, in Physicians' Desk Reference, 56th ed.
Montvale, NJ, Medical Economics Co, 2002, pp 2688-2692]
However, there is no known causal connection
between this drug and lenticular opacities.
. cataracts from chlorpromazine, thioridazine, and perphenazineHandbook of Psychiatric Drug Therapy
seem to be more frequent in young schiz'ic's
with rates in 22 ... 26%;
the young schiz'ics have an 82%
overall ocular pathology rate .
. these numbers seemed consistent with
smoking and diabetic effects
and there are multiple sources of diabetes
in most schiz'ics:
chronic stress, poor sleep, and bad diet .
. thioridazine > 800 mg results in
irreversible retinal pigmentation
which can result in blindness .
. any anticholinergic may precipitate[trazodone is weakly anticholinergic
narrow-angle glaucoma;
this happens less with the higher-potency 1st gen's,
and by avoiding co.use of antiparkinson drugs,
most of which are anticholinergics
(another class of antiparkinson drugs
is the dopamine releaser, eg: amantadine).
. thioridazine has potent anticholinergic effects .
. the antipsychotics best known for
not causing weight gain (abilify, geodon)
also may cause insomnia that leads to
co.use of benzodiazepines or trazodone;
benzodiazepines are anticholinergic .
. non-sedating antipsychotics are also[6.29:
the most likely among
later generation antipsychotics to cause
akathisia (6.29:
that is the feeling of
getting sick from having to sit still;
but moving around doesn't really help
because the lack of stimulation
has to do with the way certain meds
can affect your neuro-hormones .
people with natural akathisia
are said to have attention deficit disorder,
and the cure is amphetamines;
generally the opposite of anti-dopaminergics)
. akathisia is more likely in
the higher-powered 1st-gen's
(they have less anticholinergic properties);
so they may be co.used with
sedatives:
# beta-adrenergic blockers
(propranolol or nadolol);
# or rarely a benzodiazepine or clonidine .
. quetiapine induces cataracts in animals
so the product label susggests slit-lamp exam's;
ophthalmologic studies find no evidence
supporting regular eye exam's;
however, since schiz'ics have
other risk factors for cataracts,
a referral to an ophthalmologist
for an eye exam
can be part of routine medical care
. diabetic ketoacidosis remains a rare possiblity[ziprasidone is not a sedative;
for all these antipsychotics .
. among the new anti-aggressives,
ziprasidone and aripiprazole cause
the least weight gain,
. all schiz'ics on 2nd-generation antipsychotics[is ziprasidone 2nd generation?
should be monitored at least twice yearly
for glucose abnormalities
and lipid profile .
)my reply:. according to that article,
Dr. Loren Cordain is a professor of exercise physiology) .
and a renowned expert in the area of Paleolithic nutrition.
Call your Senators and Representative today and say
USDA must not approve GE alfalfa!
Monsanto wants to sell its genetically engineered (GE) alfalfa
and wants the USDA to approve its permit application,
but consumers, farmers, dairies, and food companies
dont want GE alfalfa plants and seeds released into the environment.
USDA's Draft Environmental Impact Statement (DEIS) admits
that if GE alfalfa is approved:
* GE Contamination of non-GE and organic alfalfa crops will occur
* GE contamination will economically impact small and family farmers
* Foreign export markets will be at risk due to rejection of GE contaminated products
* Farmers will be forced to use more toxic herbicides to remove old stands of alfalfa
Yet, unbelievably, USDA has decided that these impacts are insignificant!
And, USDA intends to approve Monsanto's Roundup Ready GE alfalfa anyway.
Call your Congressional Representatives today
and ask them to hold USDA accountable
by contacting Secretary of Agriculture Vilsack
and urging him to deny approval of Monsanto's GE alfalfa!
Tell them that you DO NOT support the deregulation of GE alfalfa,
for the following reasons:
* GE contamination of non-GE and organic crops would be inevitable
* You won't buy products that are GE-contaminated
* Alfalfa is a major food source for livestock
and GE alfalfa would destroy the integrity of organic dairy products
* You support the rights of farmers to grow the crops of their choice,
and GE contamination makes that impossible
* Ask your Representative and Senators to contact Secretary Vilsack
and urge him to deny USDA approval of Monsanto's GE alfalfa
Then email us at info@truefoodnow.org
and tell us who you called
and let us know what kind of response you got!
Background
In 2006, the Center for Food Safety (CFS)
sued the Department of Agriculture (USDA)
for its illegal approval of Monsanto's
genetically engineered (GE) Roundup Ready alfalfa.
USDA failed to conduct an environmental impact statement (EIS)
before deregulating the crop, as required by law.
An EIS is a rigorous analysis of the potential significant
environmental, health, and economic impacts of a federal decision,
mandated under the National Environmental Policy Act (NEPA).
The federal courts sided with CFS
and banned GE alfalfa until the USDA fully analyzed
the impacts of the GE plant on the environment, farmers, and the public
in an EIS.
USDA released its draft EIS on December 14, 2009.
A 75-day comment period is now open until 3 March, 2010.
CFS has begun analyzing the EIS
and it is clear that the USDA,
in its recommendation to approve GE alfalfa,
has not taken into consideration the concerns of
non-GE alfalfa farmers, dairies, exporters, retailers or consumers ...
In fact, their [current] EIS states that consumers
don't care if their organic food is GE contaminated
and neither do organic farmers,
as long as farmers employ the organic practices required
under the Organic Foods Production Act (OFPA).
Obviously, this is not true
and we need to push our Congressional representatives
to call upon USDA to deny the approval of GE alfalfa.
. review the EIS and other documents .
USDA must not approve GE alfalfa Tell-a-friend!
If you want to avoid GMO's in Canada and USA)
you need to buy organic since we don't label them here.
There are lobby groups trying to get the government to
label food products as GMO
but the companies against it have stronger lobby boards
that argue it will cause unnecessary fear among citizens.
