#Germany circumcision ban warns #jihad

10.14: news.pol/purges/prison/
circumcision ban labeled religious intolerance:
14: 19: summary:
. after England frowned on Germany for
appearing to be anti-semitic again,
I wanted to point out a misunderstanding
about Germany's issue with circumcision .
. I have German and English blood,
my family includes American Jews,
my country is both USA and UK;
my religion is supernature-aware,
but without a belief in circumcision,
and includes a love of Islam's Prophet
however I think the Prophet is misunderstood:
in this age, he would use psychology, not war,
to pursue the dream of world-wide Islam;
and I don't think he would be against Israel,
but he would insist on community purity:
neighborhoods and businesses used by muslims
should be completely Islamic .

. the news of Germany outlawing circumcision
was made into an anti-jewish spectacle,
but notice the related news just before this:
Germany's leadership was reporting
that after embracing the immigration of muslims
it looked like "(it wasn't working out) .
[citation not locatable, heard on NPR?
but below are many reports from Germany
suggesting widespread resistance to,
or systematic "integration" of, Islam .]
. indeed, that was the same time there was
serious talk of giving up all nuclear power
-- and they were concerned mostly about
having it sabotaged by terrorists .

can "(organic) include nanotech? ........... ENM's are the new GMO

7.20: co.pol/nanotech/fda needs to regulate ENM's:
Center for Food Safety office@centerforfoodsafety.org
date:     Thu, Jul 19, 2012 at 10:18 AM
subject:
    Urge FDA to Regulate Nanotechnology
    to Protect Consumer Health

The U.S. Food and Drug Administration (FDA) has a public comment period open through July 24th on the oversight of nanotechnology in food. Amazingly, the FDA currently does not regulate the use of nanotechnology in food, despite its widespread use, and serious public health concerns.
 Already a wide variety of nanoscale applications in food packaging and processing are being developed. Yet, we know very little about the health effects of exposure to engineered nanomaterials, and what we do know, is cause for alarm.
Make your voice heard and urge FDA to regulate nanotechnology to protect consumer health.
To: Office of Food Additive Safety, U.S. Food and Drug Administration (FDA)
Re: Guidance for Industry on assessment of the effects of the incorporation of Engineered Nano-scale Materials (ENMs) in food ingredients (food substances and food contact surfaces) (Docket number: FDA-2011-D-0490)
It is shocking that FDA does not yet regulate the use of ENMs in food ingredients, given that nanomaterials are reportedly widely present in food ingredients and food contact substances. A 2012 National Research Council report found that “there is little progress” on research into the effect of oral consumption of engineered nanomaterials on human health. The FDA should commission, finance and publish such research as an urgent public health priority, and a basis for regulation.
The FDA’s draft guidance to industry should be a first step towards regulation to protect public health and the environment.
Neither FDA nor industry should assume a food ingredient at its macro-scale to be safe at the nano-scale, since we know that nanomaterials can behave completely differently than their bulk counterparts. These unique behaviors are what make nanotechnology so attractive to industry, but they also raise unique risks that must be addressed through unique regulation and oversight. The FDA should require those companies to submit data showing that their products are safe. Further, these safety assessments should be publicly available and the consultation process transparent.
All engineered nanomaterials used as food ingredients, food contact substances, and food colorants should be subject to a rigorous pre-market safety assessment. FDA should withdraw from the market all products shown to contain engineered nanomaterials as food ingredients that have not undergone the pre-market safety assessment and been shown to be safe for daily human consumption.

liver-glucose reducer lowers inflammation

5.14: news.health/liver-glucose reducer lowers inflammation:

"Metformin's strong anti-inflammatory properties
by activating an enzyme called AMPK,
which in turn damps down
the activity of the protein NF-kappa B.
The inhibition of that suppresses the production of
inflammatory signaling molecules
-- cytokines and chemokines --
needed to initiate and sustain uveitis.
Uveitis causes 10 to 15 % of all blindness in the USA
and even higher proportions of blindness globally.
The only conventional treatment is steroid therapy,
which has serious side effects and cannot be used long-term.

inflammation syndrome and colon cancer

11.16: health/colon cancer

summary:
. inflammation syndrome seems to be a key;
foods tend affect risk in proportion to their inflammation index:
grain-fed meats, high-glycemic fruits
are inflammatory,
while wild fish and low-glycemic veg's
are anti-inflammatory
. aspirin too reduces both risk and inflammation,
though the dose that might be useful
might also be kidney problems .


inflammation syndrome

. conditions that contributes to inflammation of the colon can lead to cancer.
Diabetes Can Lead to Colon Cancer
According to WebMD, people with diabetes have a 30-40%
increased risk of colon cancer.
So it's important to maintain a normal weight
and to lose weight if an individual is obese.

risk-factors-colorectal-cancer

. a person who already has had colorectal cancer
may develop the disease a second time.
. those who have chronic inflammatory conditions of the colon,
such as ulcerative colitis or Crohn's disease,
also are at higher risk of developing colorectal cancer.
# Diet. A diet high in fat and calories and low in fiber

may be linked to a greater risk of developing colorectal cancer.
# Lifestyle factors.

You may be at increased risk for developing colorectal cancer if you

drink alcohol, smoke, don't get enough exercise,

and if you are overweight.
# Diabetes.

People with diabetes have a 30-40% increased risk

of developing colon cancer.

Cancer epidemiology, biomarkers & prevention

2006, vol. 15, no12, pp. 2391-2397

Obesity and diabetes are established risk factors
for colorectal cancer but have mainly been assessed independently.
There are few data about whether the metabolic syndrome,
which refers to a clustering of cardiovascular disease risk factors
thought to be related to insulin resistance,
including obesity, type 2 diabetes, hyperlipidemia, and hypertension,
is associated with colorectal cancer risk.
Out of the markers of the metabolic syndrome assessed,
overweight and diabetes are risk factors for colorectal cancer,
whereas, in contrast to their role in cardiovascular disease,
elevated blood pressure and hypercholesterolemia are not.

Smoking:

It has been shown that smokers have a 30-40% greater chance
of acquiring colon cancer than non- smokers.

http://www.aicr.org.uk/ColonCancerFAQs.stm

Obesity can increase the risk of cancer of the colon by up to one third.
High alcohol intake is also known to increase the risk of colon cancer.
However, some common drugs, such as aspirin-like painkillers
and hormone replacement therapy,
are known to reduce the risk of bowel cancer.
Low fat, high fibre diets appear to carry a lower risk.
Greater consumption of vegetables and fruit has also been shown to reduce the risk.
Increased consumption of red meat and processed meat
has been linked to a higher risk.
By comparison, eating fish does not appear to be a risk factor.
Some evidence suggests that certain dietary supplements,
such as calcium, selenium and, possibly, folic acid
can reduce the risk.

obesity from eating for stress may be another link
More Evidence Stress and Cancer are Linked Posted by: Dr. Mercola
Cancer Research November 1, 2006; 66(21): 10357-10364
Science Daily November 3, 2006

Stress

Norepinephrine, a hormone produced during periods of stress, may increase the growth rate of cancer.

The norepinephrine can stimulate tumor cells to produce two compounds (matrix metalloproteinases called MMP-2 and MMP-9) that break down the tissue around the tumor cells and allow the cells to more easily move into the bloodstream.

Once there, they can travel to other organs and tissues and form additional tumors, a process called metastasis.

Norepinephrine may also stimulate the tumor cells to release a chemical (vascular endothelial growth factor, or VEGF) that can aid in the growth of the blood vessels that feed cancer cells. This can increase the growth and spread of the cancer.
Researchers traced the harmful effect of norepinephrine after applying it to cancer cell lines used to study nasopharyngeal carcinoma (NPC), an incurable head and neck cancer associated most frequently with those of Chinese descent.